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Probing The Regulation Of Vasculogenic Mimicry In Glioblastoma: Implications For Treatment In Patients
Health and environmental sciences Biological sciences Angiogenesis Anti-angiogenic treatment resistance Cancer Cancer stem cells Glioblastoma Vasculogenic mimicry
2014/12/10
Glioblastoma, previously known as Glioblastoma Multiforme (GBM), is a highly angiogenic tumor and is defined pathologically by its ability to create microvascular proliferations. Patients with gliobla...
The Role of YKL-40 in the Progression of Glioblastoma
angiogenesis CHI3L1 GBM glioblastoma Vasculature YKL-40
2014/12/10
Glioblastoma Multiforme (GBM) is the most common brain cancer and one of the most fatal forms of cancer overall. The average survival time is 10-14 months, and less than 10% of patients survive more t...
Notch Regulation of Adam12 Expression in Glioblastoma Multiforme
microRNA mir-29 Glioblastoma Multiforme
2014/10/21
Glioblastoma is the most common malignant brain tumor, accounting for 17% of all primary brain tumors in the United States. Despite the available surgical, radiation, and chemical therapeutic options,...
Hypoxic Cell Waves around Necrotic Cores in Glioblastoma: A Biomathematical Model and its Therapeutic Implications
Glioblastoma multiforme tumor hypoxia pseudopalisades invasion mathematical model
2012/5/2
Glioblastoma is a rapidly evolving high-grade astrocytoma that is distinguished pathologically from lower grade gliomas by the presence of necrosis and microvascular hiperplasia. Necrotic areas are ty...
Association of Invasive Breast Carcinoma and Glioblastoma Multiforme: a Case Report with Histological and Immunohistochemical Features
Breast carcinoma glioblastoma multiforme immunohistochemistry associated neoplasms
2009/6/23
Breast cancer is the most common cancer (21%) and the second leading cause of cancer deaths among women. Annual breast cancer deaths are exceeded only by those from lung cancer (1). In contrast, brai...
Constitutive Expression and Inducibility of Plasminogen Activator in Mer- Glioblastoma A1235 Cell Line and in Mer+ A8 Cells Transfected with Bacterial ada Gene
A1235 human glioblastoma Mer phenotype MNNG
2009/3/23
The alkylation repair deficient (Mer- phenotype) cells produce higher levels of proteolytic enzyme plasminogen activator (PA) after treatment with alkylation agent N-methyl-N'-nitro-N-nitrosoguanidine...
Induction of Urokinase-type Plasminogen Activator by Sodium Salicylate in a Glioblastoma Cell Strain
urokinase uPA promoter sodium salicylate
2009/3/20
Urokinase-type plasminogen activator (uPA) is an extracellular protease involved in many physiological and patological processes. In this paper its induction by sodium salicylate (NaSal) in A1235 glio...
Urokinase-type Plasminogen Activator and Plasminogen Activator Inhibitor Induction by Etoposide in a Glioblastoma Cell Strain
urokinase plasminogen activator inhibitor uPA promoter
2009/3/20
Urokinase plasminogen activator (uPA) and its inhibitors (PAI) are elements of plasminogen activation system, a proteolytic system involved in many physiological and patological processes. In this pap...